What is Pathology - Respiratory Acidosis
Pathophysiology • The body's pH should range from 7.3 to 7.45. The pH is low, CO2 is high, and HCO3 is either within the normal range or increasing to make up for it, indicating respiratory acidosis according to the ABG analysis. • When the pH fluctuates, the body activates buffering mechanisms. Cellular reserves are the first to respond. In respiratory acidosis, K+ is shifted out of the cells as a result of H+ being taken into the cells. • The second buffering mechanism to engage is the lungs. Low pH causes rapid and deep respirations, which produce CO2. • The kidneys are the final buffering mechanism, and it can take them up to two days to start modifying pH. The kidneys produce H+ when there is respiratory acidosis. • The use of drugs that impair lung function, hypoventilation, sleep apnea, and COPD are some of the causes. Assessment and diagnostic findings include CXR, drug testing, polycythemia screening on the CBC, and ABG analysis. Complications include changes in mental state, cerebral edema, and papilledema as a result of chronic hypoxemia, which can also cause polycythemia, pulmonary hypertension, and cor pulmonale. Death might happen. Medical Attention and Surgical Procedure • Bronchodilators, mechanical ventilation assistance, naloxone or flumazenil for benzodiazepine and opioid overdoses, and theophylline. • Surgical procedures for treating weight and sleep apnea. • Teach the client how to maintain a healthy weight, abstain from smoking, and use oxygen as advised. • Inform the customer about available sleep study options. • Keep an eye on your ABGs, breathing, and CBC levels for polycythemia. • Determine the brain edoema score on the Glasgow Coma Scale and the presence of papilledema.
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4/27/2023 01:00:05 pm
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