What is Pathology - Venous Stasis Ulcer
Pathophysiology • Venous pressure in the lower limbs rises and blood is not returned to the heart effectively. The increased venous pressures result in waste-carrying serous fluid leaking into the interstitial area and blood flowing backward into capillary exchange beds. • Increased pressure in a vein causes a small rupture that becomes a deeper wound that cannot heal due to poor capillary access to inflammatory agents, oxygen, and glucose. • Edema in the interstitial space inhibits capillary access for all cells and can be serious. Inflammatory substances that become stuck in the subcutaneous tissue cause the wound to ulcerate by damaging the veins' valves and causing serous fluid to leak out. Evaluation and Diagnostic Results • Extremity edoema with brittle, brown skin. • The ulcer's culture was collected. • Infection from CBC to R/O. Complications • Infection, persistent ulcers that don't resolve and reduce the client's quality of life, and reulceration. • Permanent harm to the vessels' valves. Medical Attention and Surgical Procedure Unna's thigh is elevated as the compression bandage on her boot is wrapped. • The use of pentoxifylline. • Skin transplants and synthetic skin grown in culture. • Clients are told to stroll if they can. • Stress the value of lifting the thighs while walking and using the skeletal muscle pump. • Check for recovery after removing the compression boots. (every 2–7 days). • The region should not be disturbed if skin grafts are used until skin buds are visible.
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