What is Pathology - Ventricular Fibrillation
Pathophysiology • CAD, MI, and structural or inflammatory cardiac diseases are linked to VF. Administration of antiarrhythmic medications, atrial arrhythmia, cardioversion, and hypoxic conditions can all cause it. • Marfan's syndrome, tetralogy of Fallot, Kawasaki's disease, long QT syndrome, and Wolff-Parkinson-White syndrome are congenital diseases that predispose to VF. • Hyperkalemia and hypomagnesemia, cardiac catheterization, and pacemaker wire placement are other causes of VF. Evaluation and Diagnostic Results • The ECG exhibits an erratic beat and lacks a P wave, PR interval, or QRS complex. • The client instantly loses consciousness, has no pulse, and no blood pressure. • Cyanosis, respiratory failure, cardiovascular collapse, and pupillary enlargement take place. Death, cerebral anoxia, aspiration pneumonia, defibrillation injury, and recurrent VF events are all complications. Medical Attention and Surgical Procedure • Following the ACLS procedure, immediate defibrillation, oxygen therapy, intubation, and the administration of antiarrhythmics, anticholinergics, and vasopressors. • CABG to treat circulation issues. • ICD for individuals at recognised risk. • Promote the use of AEDs and CPR among all clients and family members for personal protection and the advancement of public health. • Inform clients that if they feel any chest discomfort, they should call emergency services. • Adhere to the ACLS procedures and algorithms to improve the chances of the client surviving. Defibrillate right away, and constantly keep an eye on your vital signs, oxygen saturation, and ECG. • Adhere to pacemaker safety procedures
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